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| DC 欄位 | 值 | 語言 |
|---|---|---|
| dc.contributor.author | 孫斐 | zh_TW |
| dc.date.accessioned | 2021-07-01T08:15:10Z | - |
| dc.date.available | 2021-07-01T08:15:10Z | - |
| dc.date.issued | 1990 | |
| dc.identifier.citation | 杜啟安.1980.血液學技術.黎明書店,新竹.臺灣.498pp.
孫 斐.1987.幼鯉魚檢定急性魚毒-測試液更新法.臺灣省農業藥物毒物試驗所應用毒理系技術專刊第四號. 臺灣區農藥工業同業公會.1988.臺灣區農藥工業同業公會七十七年度會員廠農藥產銷統計.臺灣區農藥工業同業公會,臺灣省.63pp. Abo-Khatwa, N. and R. M. Hollingworth. 1974. Pesticidal chemicals affecting some energy-linked functions of rat liver mitochondria in vitro. Bull. Environ. Contam. Toxicol. 12: 446-454. Adams, S. M. and R. B. McLean. 1985. Estimation of largemouth bass, Micropterus salmoides Lacepede, growth using the liver somatic index and physiological variables. J. Fish Biol. 26: 111-126. Baker, E. M. 1967. Vitamin C requirements in stress. Am. J. Clin. Nutr. 6: 583-590. Bardach, J. E., M. Fujiya and A. Holl. 1965. Detergents: effects on the chemical senses of the fish Ictalurus natalis (Ie Sueur). Sceince. 148:1605-1607. Bengtsson, B.-E. and A. Larsson. 1986. Vertebral deformities and physiological effects in fourhorn sculpin (Myoxoce-Phalus guadricornis) after long-term exposure to a simulated heavy metal-containing effluent. Aquat. Toxicol. 9: 215-229. Bradshaw, J. S., E. L. 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In vitro metabolism of pyridine and chlorpyrifos by rainbow trout liver nicrosomes Master thesis, Oregon State University, Oregon, USA Zannoni, V. G. and P. H. Sato. 1975. Effects of ascorbic acid on microsomal drug metabolism. Ann. N. W. Acad. Sci. 258: 119-131. | |
| dc.identifier.uri | http://tdr.lib.ntu.edu.tw/jspui/handle/123456789/75758 | - |
| dc.description.abstract | 吳郭魚受陶斯松的影響,暴露在70μg/l中30天後,累積死亡率達43%,並有15%骨骼發生變形、生長受阻,肝-體指數(LSI)顯示在暴露5天後魚體肝的狀況與對照組有差異存在;魚腦和肌肉中乙醯膽鹼酯?(AChE)的活性,則在暴露3小時內即已降低50%以上;腦和肝臟中貯藏的維生素C (AsA)、肝和肌肉中貯存的總蛋白質含量均顯著降低,顯示70μg/l的陶斯松會誘導魚體內AsA和能量的大量消耗,AChE的活性受抑制、骨骼變形的發生,可能也與AsA的消耗有關;病理切片結果証實,在暴露5天後,肝有萎縮壞死的現象;血液學上的分析並顯示有貧血的情形. 暴露在21μg/l中的吳郭魚,30天內累積死亡率為27%、骨骼變形發生率為4.6%,比70μg/l為低,吳郭魚的生長雖然沒有明顯的受到抑制,但健康係數(CF)和LSI值在30天時,比對照組顯著降低,顯示魚的健康和肝的狀況均不佳.其他生理生化上所受的影響,與暴露在70μg/l中的魚相似. 暴露在11μg/l中的魚,累積死亡率為6.3%、骨骼變形者佔2.1%,生長雖然未受阻,但30天後魚體和肝的狀況仍與對照組有差異,AChE活性受抑制達50%以上需時1日,但腦和肝中AsA的含量並沒有明顯的降低,暗示仍有生合成的能力.暴露30天後,肝細胞也出現萎縮和壞死的現象,血液中血球容積比雖然受到影響,但血紅素與紅血球數目與對照組無顯著的差異. 綜合上述,陶斯松對吳郭魚生理生化上的影響與暴露的時間和濃度有關,暴露在高濃度中的魚,經較短的時間,暴露在低濃度中的魚,經過較長的一段時間後,生理或生化上均發生改變,但高濃度與低濃度對吳郭魚所造成的影響不同,尤其是環境壓力導致魚體對維生素C的消耗以及生合成的能力上有差異. | zh_TW |
| dc.description.provenance | Made available in DSpace on 2021-07-01T08:15:10Z (GMT). No. of bitstreams: 0 Previous issue date: 1990 | en |
| dc.description.tableofcontents | 壹、緒言……………………………………1 貳、前人研究……………………………………5 三、材料與方法……………………………………8 一、測試藥劑……………………………………8 二、試驗動物……………………………………8 三、實驗設計……………………………………8 四、水中陶斯松殘留量分析……………………………………9 1.分析方法……………………………………9 2.使用儀器及條件……………………………………10 3.測定及計算……………………………………10 4.回收率試驗……………………………………10 五、各種生理生化改變的測定方法……………………………………11 1.生理學上的技術……………………………………11 2.生化分析……………………………………11 3.病理切片技術……………………………………14 4.血液分析技術……………………………………15 六、結果分析……………………………………15 肆、結果……………………………………16 一、陶斯松在水中的殘留量……………………………………16 二、魚體形態的改變……………………………………16 三、對魚體內乙醯膽鹼脂?活性的影響……………………………………17 四、對魚體內維生素C含量的影響……………………………………18 五、對魚體內總蛋白質和肝糖含量的影響……………………………………19 六、病理解剖……………………………………20 七、對魚血液的影響……………………………………21 伍、討論……………………………………22 陸、參考文獻……………………………………28 柒、圖表……………………………………43 | |
| dc.language.iso | zh-TW | |
| dc.title | 不同濃度之陶斯松對吳郭魚(Oreochromis mossambicus)的影響 | zh_TW |
| dc.date.schoolyear | 78-2 | |
| dc.description.degree | 碩士 | |
| dc.relation.page | 76 | |
| dc.rights.note | 未授權 | |
| dc.contributor.author-dept | 生命科學院 | zh_TW |
| dc.contributor.author-dept | 動物學研究所 | zh_TW |
| 顯示於系所單位: | 動物學研究所 | |
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